XiaoMi-AI文件搜索系统
World File Search System7r
基于地理空间云框架的可持续农业大数据分析
人类面临着巨大的挑战：如何提高农业产量，实现 21 世纪的粮食安全，养活预计将增长到 100 亿的人口。这需要在保持可持续农业系统的同时，应对气候变化、水资源枯竭以及极端天气事件导致的土壤侵蚀和生产力下降等挑战。精准农业诞生于 20 世纪 80 年代，得益于 GPS 和卫星图像等几项关键技术的发展。本文认为，随着气候变化的影响日益加剧，精准农业和农业的下一次革命将由可持续精准农业和环境 (SPAE，类似于 7R) 驱动，它可以利用过去的技术与大数据分析相结合。这种新的以技术为中心的 SPAE 从特定地点的管理重点转变为全球可持续性的概念。为了实现这一转变，我们引入了 WebGIS 框架作为组织原则，将本地、特定地点的数据生成器（称为智能农场）与区域和全球农业视图联系起来，从而为农业行业和政府决策者提供支持。这将有助于将位于网络中的数据库集成到一个系统中，以实现所需的 SPAE 管理和连接
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双特异性免疫细胞参与者在体外增强了CD16+ NK细胞和巨噬细胞的抗癌活性，并消除了NK人源化NOG小鼠的癌症转移
抽象背景在肿瘤微环境中发现了多种类型的产生IL-17的免疫细胞。然而，它们在CD8 +肿瘤浸润淋巴细胞（TILS）的肿瘤进展和精疲力尽中的作用尚不清楚。确定17型免疫力在肿瘤中的作用的方法，我们研究了B16F10黑色素瘤的生长以及CD8 + TIL在IL17A - / - 小鼠中的耗尽，IL17A CRE R26 DTA小鼠，RORγT抑制剂造成的小鼠，或其相应的对照组。在B16F10的先天小鼠中进行了肿瘤特异性IL-17产生的T细胞的过继转移。抗CD4或抗Ly6g抗体分别用来耗尽CD4 + T细胞或CD11b + Gr-1 HI髓样细胞的体内。通过询问TCGA数据集，评估了人类癌症中17型免疫力与T细胞衰竭之间的相关性。结果CD4 + T细胞的耗竭促进了CD8 + T细胞的耗尽，而肿瘤中产生IL-17的CD8 + T（TC17）细胞的耗尽。与产生CD8 + T（TC1）细胞不同，肿瘤浸润的TC17细胞表现出CD103 + KLRG1-IL-7RαHIHI组织驻留的记忆样表型，并且细胞溶解度不佳。产生IL-17的肿瘤特异性T细胞的产物转移增加，而产生IL-17的细胞的耗竭降低，肿瘤中PD-1 HI TIM3 + TOX +终末耗尽的CD8 + T细胞的频率。封锁IL-17或RORγT途径抑制CD8 + T细胞的耗尽，还会延迟体内肿瘤的生长。封锁IL-17或RORγT途径抑制CD8 + T细胞的耗尽，还会延迟体内肿瘤的生长。与这些结果一致，人类TCGA分析揭示了多种癌症中17型和CD8 + T细胞耗尽签名基因集之间存在很强的正相关。结论IL-17产生的细胞促进了CD8 + T细胞的末端衰竭和体内肿瘤进展，可以通过IL-17或RORγT途径的阻断来逆转。这些发现揭示了IL-17-产生细胞作为促进CD8 + T细胞耗尽的肿瘤细胞的新作用，并提出17型免疫力作为癌症免疫疗法的有希望的靶标。
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